CD34 and myocardial infarction: Given the emerging role of epigenetic phenomena as fundamental triggers of (stem) cell differentiation and plasticity, in the present study we studied whether modification of the epigenetic landscape by pharmacologic inhibition of histone deacetylases (HDAC) enzymes affects CD34+ cells growth, stemness, phenotype and gene expression; we also assessed whether HDACi-treated CD34+ cells have a modified or enhanced regeneration capacity in a mouse model of myocardial infarction.