In addition to promoting a migratory and invasive phenotype, EMT may contribute to patient mortality by conferring paclitaxel resistance in epithelial ovarian cancer cells [69], inducing myocyte differentiation into CD14+/KDR+ proangiogenic cells thereby promoting tumor angiogenesis and vascularization [70], and inducing the differentiation of stromal mesenchymal stem cells into tumor associated fibroblasts that support disease progression through matrix remodeling [41]. The gene discussed is KDR; the disease is neoplasm.