Many of these mRNAs were gradually reduced in transition from normal to recruited and to tumor states, correlating with loss of oligodendroglial character (Aspa, Mobp, Tfr, Mog), evasion of cell death (Mal, Ndrg1, Gjb6, Gpr37), maintenance of undifferentiated phenotype (Mal, Ndrg1, Efhd1, Kndc1, Ermn), cell-cell and cell-matrix interactions (Thbs3, Tppp3, Itih3), and cell signaling (Ppp1r14a, Gja1). The gene discussed is ITIH3; the disease is neoplasm.