Nevertheless, it did not reflect on other metabolic factors such as blood glucose or glucose tolerance - the IGF-II/LDLR-/-ApoB100/100 mice showed both fasting hyperglycemia (8.2 ± 3.0 mmol/l) and impaired glucose tolerance in intraperitoneal GTT (2-hour post-glucose load 14.7 ± 6.0 mmol/l) compared to LDLR-/-ApoB100/100 and C57Bl/6J mice (Table 1 Figure 1). The gene discussed is LDLR; the disease is Hyperglycemia.