SNORD24 and multiple sclerosis: U24, a membrane glycoprotein from Human Herpesvirus Type-6A (HHV-6A), has garnered recent interest because a N-terminal fragment of the protein was shown by Tejada-Simon et al. to activate T-cells [1], and cause them to cross-react with myelin basic protein, an autoantigen targeted in the pathogenesis of multiple sclerosis (MS).