Earlier, we reported that a progressive decline in myocardial performance at 3 and 7 days in a hyperdynamic model of sepsis is associated with increased levels of proapototic caspase-3, increased B-cell leukemia (Bcl2)-associated protein×(Bax)/Bcl2 ratio and release of cytochrome C [8]. This evidence concerns the gene CYCS and Sepsis.