The possible scenario of molecular events in cervical cancer suggested by our study portrays the evasion of apoptosis mediated in two ways, first, the sequestration of the pro-apoptotic protein BAD by 14-3-3ζ [60] and the overexpression of the anti-apoptotic protein Bcl-2 managed by the interaction of c-Myc and E2F1 [61]. This evidence concerns the gene BCL2 and cervical cancer.