As described previously, this could be due to retention of transcription factors that are lost during the development of other types of breast cancers, or related to a specific translocation others have described in secretory carcinomas, t(12; 15) (Tognan et al. 2002), that results in the fusion protein ETV6-Ntrk3 possibly activating STAT5a. This evidence concerns the gene ETV6 and breast carcinoma.