KRAS and adenocarcinoma: To further exclude the possibility that the regulated phosphorylation resulted from differential expression of these proteins, we analyzed the expression status of the corresponding genes in a microarray dataset (NCBI's GEO database, Reference GSE11969) related to clinical samples, where normal lung (n = 5) and adenocarcinoma samples with (n = 12) and without (n = 84) KRAS mutations were compared.