For this we studieddifferent models of psoriasis, such as the imiquimod-induced psoriasis-like skininflammation in mice [22] and ex vivo-stimulated human skinexplants, and investigated GATA3 expression and activation in keratinocytes duringepidermal regeneration, which is known to share many characteristics with psoriaticskin inflammation [23]. The gene discussed is GATA3; the disease is psoriasis.