The finding on kidney correlates MT as a biomarker of glomerular kidney dysfunction by dose-response relationships between urinary-Cd and the prevalence of increased levels of biomarkers in urine of renal tubular dysfunction such as urinary beta-2-microglobulin or N-acetyl-beta-D-glucosaminidase - NAG or urinary albumin [13]. The gene discussed is OGA; the disease is renal tubule disorder.