Tables 2 and 3 show the prevalence and median levels of cytoadherence phenotypes respectively. Although not statistically significant, intensities of adhesion to CD36 were always lower in isolates from children with severe malaria than in their matched controls (Table 3). Prevalence and level of platelet-mediated clumping was significantly higher in isolates from severe cases compared to matched controls, both when the assays were conducted with unadjusted parasitemias (Table 2 and 3) and when parasitemias were adjusted to 1% (Table S1). This evidence concerns the gene CD36 and parasitic infectious disease.