IGKV2D-24 and non-small cell lung carcinoma: The mitogenic effects of nicotine were abrogated by α7 subunit antagonists, α-bungarotoxin, and methylallyl aconitine (MAA), whereas it was unaffected by α-lobeline (α4β2 subunit inhibitor) or dihydro β-erythoidine (DHβE; α3β2 and α4β2 subunit inhibitor), suggesting that α7 subunits primarily mediated the mitogenic effects of nicotine in NSCLC cells.