IFNGR1 and infection: Three weeks after de novo infection, we found that surface expression of ICAM-1, MHC-class I, HLA-E, IFN-γR1 and NKG2D-ligands by both GFP– and GFP+ KSHV-infected cells in established persistent latency was the same as in uninfected controls, consistent with a lack of modulation of NK-cell cytotoxicity.