NGF and chronic myelogenous leukemia, BCR-ABL1 positive: We have previously shown that NGF-TrkA signaling partially rescues TrkA expressing Bcr-Abl transformed chronic myelogenous leukemia (CML) cells, such as K562, and Meg-01, from cell death induced by a potent inhibitor of Bcr-Abl tyrosine kinase, imatinib mesylate (imatinib).