IL1RL1 and inflammatory response: Unlike in ST2-deficient mice [33], [34], [35], the development of OVA-induced airway inflammation was aggravated in mice injected with ST2-deficient OVA-specific TCR (DO11.10)-expressing Th2 cells in comparison with those injected with wild-type DO11.10 Th2 cells after OVA challenge [34].