Rapid activation of resident inflammatory cells (mostly microglia), productions of inflammatory cytokines such as interlerukin10 (IL-10) and tumor necrosis factor α (TNF-α) and translocation of intercellular transcription factors such as nuclear factor-kappa B (NF-κB) are characters of local inflammatory responses to ischemia in brain [7–11]. Here, NFKB1 is linked to ischemia.