This is supported in fixed dystonia by our recent data6 and in “pure” CRPS1, where mental rotation abnormalities that correlate with duration of symptoms and perceived intensity of pain are found.23 With regard to dystonia, mental rotation abnormalities have also been reported in DYT1 mutation carriers (both manifesting and nonmanifesting24) and primary hand dystonia,8 and therefore this abnormality is not specific to fixed dystonia. The gene discussed is TOR1A; the disease is Dystonia.