The downregulation of miR-29 and upregulation of its oncogenic targets, Tcl1 (T-cell leukemia/lymphoma 1), Mcl1 (an anti-apoptotic Bcl-2 family member) and DNA methyltransferase (DNMT3), have been implicated in chronic lymphocytic leukemia, cholangio-carcinoma and lung cancer as a means of blocking tumor cell apoptosis and silencing tumor suppressor genes [31,32]. Here, MCL1 is linked to neoplasm.