Our own bioinformatics analysis of overexpression of CIP2A and components of EGFR-MEK1/2-ETS1 pathway revealed M6 subtype of acute myeloid leukemia (AML) as a cancer type in which CIP2A and representative genes of each level of the pathway are significantly up regulated (Fig. 8) [31], [32]. This evidence concerns the gene EGFR and acute myeloid leukemia.