Given the variation in the CAG repeat length of the androgen receptor (AR) gene and its inverse effect on the receptor activity, the alleles with longer CAG repeat length are expected to result in diminished androgen receptor activity, which might lead to a state of biochemical hyperandrogenism, i.e. increased serum levels of androgens as demonstrated in a couple of previous studies [5], [7]. This evidence concerns the gene AR and hyperandrogenism.