These molecules induce the selective degradation of ∆N-p73 mRNA variants; down-regulation of aberrantly expressed ΔN-p73 isoforms in neoplastic cells as well in rapidly growing tumor xenografts resulted in inhibition of tumor growth and sensitization to chemotherapy, supporting the further development of ΔN-p73 inhibitors as potentially new anticancer agents [169]. Here, TP73 is linked to neoplasm.