SCGB3A2 and congenital rubella syndrome: As shown in our current study, SCGB3A2 (UGRP1) expression could be modulated by proinflammatory, Th1 and Th2 cytokines in nasal mucosa; therefore, the decreased expression of SCGB3A2 (UGRP1) in CRS might relate to the specific local cytokine environment in CRS [5,25].