KIR2DL3 and incontinentia pigmenti: The trend seen with IP infants having higher representation of homozygosity for KIR2DL3 and C2 compared to EU infants was substantially more significant post MVL adjustment and also appears to be apparent under conditions of low maternal viral load (Table 2), again highlighting a role for KIR2DL3, in the absence of its ligand, with increased infant IP-susceptibility to HIV-1.