In PD patients, iron deposition near degenerating neurons [169], could be intensified by hereditary mutations in iron regulatory binding proteins [170,171], iron storage/transport proteins such as ferritin (L/H subunits stabilization: storage/ferroxidase mediated uptake and utilization), caeruloplasmin, iron regulatory protein 2, lactoferrin/melanotransferrin receptors or the divalent metal transporter-1 [172,173]. Here, SLC11A2 is linked to Parkinson disease.