In addition, we found significant overlaps with genes over-expressed as a result of activities of Mutant p53, MLL and ALL, and with the loss of activity of Estrogen Receptor, TEL-AML translocation, B-Catenin, ERBB2 and other "potential regulator genes" that are found in Table S1, and summarized in Table 2. Here, TP53 is linked to acute myeloid leukemia.