IL10 and extrapulmonary tuberculosis: Mutations in P2X7, [22] vitamin D receptor, [23] interleukin (IL) 1 - β, [24] LTA,[25] IL-10 [25], and IL-10 together with IFN-γ,[26-29] have been associated with extrapulmonary disease in previous candidate gene studies, and support the hypothesis of a genetic predisposition to extrapulmonary tuberculosis.