The expression of maturation surface markers, including CD40, CD80, CD83 and MHCIwas up-regulated in UV-P3-stimulated, but not in P3-infected bmDCs and spDCs or mock-treated DCs (Figure 2), indicating that UV-P3 stimulation accelerated the maturity of DCs whereas P3 infection dramatically inhibited the cell maturation process. The gene discussed is CD40; the disease is infection.