Studies with Dectin-1-deficient mice have demonstrated Dectin-1's protective role during infection with Candida albicans6, Aspergillus fumigatus7 and Pneumocystis carinii8; however, these studies have also highlighted some inconsistencies with regards to Dectin-1's role in the control of infection with C. albicans, with both redundant and non-redundant roles observed 6, 8. This evidence concerns the gene CLEC7A and infection.