CCR4 and systemic lupus erythematosus: Various disease models such as experimental autoimmune encephalomyelitis (EAE), systemic lupus erythematosus (SLE), or nephrotoxic glomerulonephritis (NTN) have been linked to infiltration of Th17 cells, most of them reporting CCR6 as a key factor for Th17 infiltration into the target tissues as well as a potential role for CCR4-directed migration [104, 108–112].