LRP8 and autoimmune polyendocrinopathy: Toll-like receptors, annexin A2, ApoE receptor (ApoER2), platelet receptor GPIb and anionic phospholipids exposed on cellular surfaces of activated cells are suggested to be pathologically important in APS [18], [19], [20], [21], [22], [23], [24], [25], [26], [27], [28], [29].