That IFNs might be potential anticancer drugs led to widespread, unwarranted, and later disappointed expectations of their being a general cure for cancer; on the positive side, however, interest in finding better sources for a potential wonder drug led directly to the cloning of genes for human IFN-α [12], and later for IFNs-β and -γ [13, 14]. Here, IFNA1 is linked to cancer.