Among these presumptions, a viral infection has been accepted, as supported by the demonstration of local intraocular antiviral antibody production [16-18],Elevatedγ-interferon and interleukin 10, an increased number of CD8+ T cells in the aqueous humor and a positive cellular response to retinal S antigen have been reported in patients with Fuchs’ syndrome [19-22]. Here, CD8A is linked to viral infectious disease.