When further dividing the H. pylori infected by virulence (Table 3), no significant differences in gastric cancer risk were observed (e.g., highest vs lowest third of sodium intake from FFQ: OR=4.50, 95% CI: 0.18–112.64 among the H. pylori-negative group, and OR=3.93, 95% CI: 0.30–52.27 in the H. pylori-positive/CagA-negative group, P for interaction=0.612; and OR=1.52, 95% CI: 0.71–3.28 in the H. pylori-positive/CagA-positive group, P for interaction=0.407). The gene discussed is S100A8; the disease is gastric cancer.