TGFB1 and cytomegalovirus infection: These data provide supportive evidence that HCMV infected renal epithelium may contribute to long-term renal allograft loss by exacerbating fibrosis via TGF-β1, and provides a potential in vitro mechanism for the association of HCMV infection with greater TGF-β1 production in renal allografts and long-term allograft loss.