Elucidation of factors that contribute to the infiltration of immune cells into the ovarian cancer microenvironment (but not breast cancer) [108–110], revealed that tumors with significant T cell infiltrates had elevated levels of various chemokines, including CCL5, the production of which was found to be restricted to the lymphocyte population rather than the tumor cells [111, 112]. This evidence concerns the gene CCL5 and neoplasm.