An alteration in peroxisome proliferator-activated receptors (PPARs) has been described in common complications of pregnancy, including GDM, intrauterine growth restriction (IUGR), and preeclampsia (PE) [86–88], which suggests the existence of a similar epigenetic control of the PPAR or PPARGC1A genes, as has been described for T2D. Here, PPARA is linked to fetal growth restriction.