FCGR3B and rheumatoid arthritis: Furthermore, the FCGR3B#1 probe reported homozygous deletion (0 copies) in 16.9% of healthy controls versus 12.9% RA patients, and gene amplification (3 copies) in 0.4% of healthy controls versus 2.4% of RA patients (OR = 6.23, P = 0.05).