Interestingly, other non-mitochondrial proteins significantly upregulated in Moraxella-positive kidney tissue were peroxiredoxins and S-adenosyl-homocysteine hydrolase, together with indications about upregulation of antiquitin, Warm acclimation-related protein 65, transferrin, glutathione S-transferase, carbonic anhydrase, and Cu/Zn superoxide dismutase, many of which are known to be related to cellular responses to oxidative stress, infection, inflammation, or programmed cell death processes. The gene discussed is SOD1; the disease is infection.