Since the above results suggest that GSK-3α can antagonize BZ-dependent cell death and as it has been demonstrated that GSK-3 serine and tyrosine phosphorylation levels and intracellular localization change upon different apoptotic noxae [48], we checked GSK-3 phosphorylation and cellular localization in MM cells upon treatment with BZ. Here, GSK3A is linked to Miyoshi myopathy.