These studies demonstrated that p120 mediated inflammatory responses in the skin [21], that salivary gland development was dysmorphic and that acinar cell development was blocked [19], that spine and synapse densities along dendrites were dramatically reduced [20], that p120 regulates keratinocyte mitosis and its absence promotes tumor formation [27], and that p120 enhances endothelial proliferation and periocyte coverage of developing microvessels [40]. This evidence concerns the gene CTNND1 and neoplasm.