Studies by other laboratories support this finding; NButGT, unlike PUGNAc, does not hinder activation of Akt in cultured astrocytes (Matthews et al., 2007) and NButGT does not exacerbate high glucose-induced insulin resistance in L6 myotubes (Srinivasan et al., 2009). The gene discussed is AKT1; the disease is Insulin resistance.