ELN and leptospirosis: Later during dissemination, the ability of OmpL37 to efficiently bind human aorta elastin suggests that OmpL37 could promote leptospiral attachment to elastin-rich vascular structures, including the walls of the pulmonary, cardiac, and other blood vessels, possibly explaining the propensity of leptospirosis to result in hemorrhagic complications [1], [50], [51].