IFNG and viral infectious disease: This suggests that the BGM model imposes a more severe penalty for complexity (non-stationarity), which increases with increasing sample size m. This tendency may explain the finding in [4] for the macrophage gene expression time series, which we have reproduced in the present study (Figure 3(c)): the BGM model does not infer a clear two-phase nature of the time series under simultaneous immune activation (with IFNγ) and viral infection (with CMV).