In this regard, it has been recently published that, in colorectal cancer cell lines, mutations in KRAS (v-Ki-ras2 Kirsten rat sarcoma viral oncogene homolog) or BRAF (v-raf murine sarcoma viral oncogene homolog B1) genes, are able to trigger an overexpression of GLUT1 and an increase of the glucose uptake. The gene discussed is BRAF; the disease is colorectal cancer.