TMEM37 and chronic myelogenous leukemia, BCR-ABL1 positive: Combined with previous studies showing PR1-CTL kill CML and contribute to CR in IFN-sensitive CML patients [16], our current study suggests that PR1-CTL expand during IFN therapy and that IFN may facilitate long-term persistence of PR1-CTL in a subset of CML patients that remain disease-free after all treatment has stopped.