Although that observation is consistent with a previous notion that self-reactive CD4 T cells in non-diabetic subjects might confer protection by producing regulatory cytokines such as IL10 while those cells in diabetic groups could not [9], the average scores (−log[p] = 0.4483±0.1609 for non-T1D group vs. 0.04909±0.04157 for T1D group) for both populations were lower than our 95% confidence limit. The gene discussed is CD4; the disease is type 1 diabetes mellitus.