Therefore increased expression of the surface markers of mDCs with a positive correlation between CD11c, CD86, and MHC Class II molecules and the number of ectasic vessels observed in the present study may support the concept that a more severe and extensive chronic inflammation modulated by DCs takes place in the coronary circulation of CAD patients with CAE in correlation with the widespread involvement of CAE. The gene discussed is CD86; the disease is coronary artery disorder.