BNIP3 and scleroderma: Thus several molecular functions repressed in CECs of scleroderma patients, were restored after iloprost infusion as shown by the over-expression of antiapoptotic genes (RASA1, PRKAA1 and BNIP3) and of the transcripts encoding for adhesion molecules (ICAM1, ITGA3, ITGA9), chemokines and wound healing process (EREG, CTGF).