Similarly, studies in Retnla-deficient mice showed that absence of resistin-like molecule alpha (Retnla/FIZZ1) exacerbated Th2 cytokine responses in S. mansoni-egg-induced lung inflammation, suggesting that Retnla/FIZZ1 is able to control effector Th2 responses in S. mansoni egg-induced inflammation and infection [29], [30]. Here, RETNLB is linked to infection.